Due to the high clinical variability in presentation of Déjerine-Roussy syndrome, it is impossible to predict which patients with a thalamic stroke will develop pain. Stroke and pain can sometimes result in Dejerine-Roussy syndrome. Available treatments include antidepressants, anticonvulsants, and. Improvement in neurological signs and symptoms of thalamic syndrome ( Dejerine-Roussy Syndrome) due to a stroke 20 years previously (in.

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Some affected individuals may experience short bursts of sharp, excruciating pain, which has been compared to the pain that occurs when a dental probe strikes an exposed nerve. It is found to riussy effective, but more trials are needed.

Anticonvulsants If antidepressants are not effective, an anticonvulsant like carbamazepine is added, especially if the pain is sharp and lancinating. As of the early s, longer treatments lasting months to years have been explored in the continued search for permanent removal of abnormal pain. Rousy S, Serpell M. Together we are strong.

Check for errors and try again. The syndrome was named after them after their deaths. For information about clinical trials conducted in Europe, contact: Springer Verlag an imprint of SpringerNature dejrine, Poliomyelitis Demyelinating disease Transverse myelitis Tropical spastic paraparesis Epidural abscess. Although FDA approved for chronic pain, unbiased assessment of the entire database shows that use of ziconotide has a very low therapeutic index with lack of consistent benefit in a majority of patients.


Dejerine J, Roussy G. Therefore, damage to the left hemisphere can cause both an indifference to pain and hypersensitivity to pain dysaesthesia or allodyniawhile damage to the right hemisphere can cause denial as a defense mechanism anosognosia and somatoparaphrenia.

Pain can dejerinr during the day and can be affected by several factors including touch, emotions such as stress, certain movements or overall level of activity, and temperature changes, especially cold temperatures.

Amitriptyline is an antidepressant. The location of the pain also varies considerably, and can affect a large part of the contralateral body or a smaller portion depending on the exact size and location of the thalamic lesion The incidence of venous thromboembolism following stroke and its risk factors in eastern China. However, drug therapy remains the first-line therapy for most affected individuals.

The paddle is inserted through two burr holes or via the temporary removal of a small piece of the skull craniotomy.

Dé​jerine-Roussy syndrome | Radiology Reference Article |

Clinical and sensory examination to map the sensory abnormality and to rule out the other causes of pain. Symptoms are generally not treatable with ordinary analgesics.

The thalamus is generally believed to relay sensory information between a variety of subcortical areas and the cerebral cortex.


Central post-stroke pain is a kind of hemidystonia that typically involves the areas of the dejedine affected by stroke.

Dejerine-Roussy Syndrome | Edward Tobinick MD

Symptoms of peripheral neuropathic pain e. Transcutaneous electrical nerve stimulation TENS: Retrieved from ” https: Dysaesthesia is defined as pain due to thalamic lesioning. Diagnosis and assessment of neuropathic pain. Most frequently the symptoms start within the first 6 months but can develop after 10 years of infarction.

Dejerine–Roussy syndrome

Intracerebral hemorrhage can precede central post-stroke pain in the legs because of the pattern of arrangement of fibers to face, arm, trunk, and leg is mediolateral, in the ventral posterior nucleus of the thalamus. Similar articles in PubMed.

Many chemical medications have been used for a broad range of neuropathic pain including Dejerine—Roussy syndrome. People with this disorder may be extra-sensitive or have a heightened response to acute painful stimuli hyperalgesia, hyperpathiawhich means that pain that would normally be small or minimal is felt to a far greater degree.

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